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Author: N. Gambaryan, S. Cohen-Kaminsky, D. Montani, et al Date Published: January-2012 Source: European Respiratory Journal
We read with great interest the recent editorial by Stenmark et al. [1] about the potential role of fibrocytes in pulmonary arterial hypertension (PAH) and the original article by Nikam et al. [2] showing the contribution of bone marrow-derived circulating fibrocytes to hypoxic vascular remodelling and inhibition of their recruitment by treprostinil.
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